Where are your nodules located? Is there any sign of a cord? This isn't a "big deal". You have nothing to do right now if it is Dupuytren's except monitor closely. There is a window of opportunity for RT (see above in "Treatments") while nodules are active and before contraction.

I have 3 nodules and cords in my right palm and 1 nodule and cord on my left palm. I have no contractions, but the fingers connected to the cords are stiff, and won't push backwards. Both hands ache, not specifically where the nodules are, but are very sensitive and painful on the top of my hands at times.

I am game to wait and see what happens, but if I do have DD I think NA or RT should be done before a year goes past. I'll continue to monitor what and if more changes happen and compare the wrist that received the cortisone injection to that wrist that didn't.

My appointment with my third hand-specialist orthopedic doctor was very interesting. He told me he thought I had
Non-Dupuytren’s Disease of the Palmar Fascia. He said I needed to wait long to see what developed before he could recommend any treatment (if ever.) Below is the info in an abstract he sent me.


Abstract

The typical Dupuytren’s disease patient is of Northern European descent with bilateral progressive multiple digital contractures and is genetically predisposed, with a family history. Palmar fascial proliferations sometimes present as a different entity without the typical Dupuytren’s disease characteristics. We identified 39 patients (20 women and 19 men) over a 4-year period with “Non-Dupuytren’s palmar fascial disease”, with unilateral involvement, without family history or ectopic manifestations. Twenty-three patients presented with unrelated complaints and were discovered, incidentally, to have the condition. In 28 patients, prior ipsilateral hand surgery or trauma precipitated the condition. Other related factors were diabetes mellitus and cardiovascular disease. Ten patients had skin tethering and subcutaneous thickening akin to Dupuytren’s nodules and 29 had palmar fascial thickening into ill-defined pretendinous cords. The diseased tissue was in the line of the ring finger in 30 patients. The time from insult to onset of contracture averaged 3.6 months and from onset to follow-up averaged 5.3 years. The condition was non-progressive, or partially regressive, in 33 patients. Seven patients had operations for unrelated conditions and underwent simultaneous fasciectomy without recurrence. Environmental factors, especially trauma, surgery and diabetes, are important in the pathogenesis of Non-Dupuytren’s palmar fascial disease, but these patients do not appear to be genetically predisposed for Dupuytren’s disease. Typical Dupuytren’s disease and Non-Dupuytren’s palmar fascial disease are two clinical entities that run different courses and do not share a similar prognosis. This should be taken into account in future epidemiological and outcome studies.